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江苏医药编辑部: 读了贵刊1980年第2期“读者、作者、编者”栏刊登的南京医学院胡介堂医师关于“磷化锌在人体内的作用过程及如何抢救”的问题解答,受益非浅。正如作者所指出的,口服磷化锌后临床主要表现为口腔粘膜和咽喉糜烂、口干舌燥、腹部不适、说话无音,恶心呕吐腹泻、肝肿大、黄疽、肝区疼痛、全身出血,白细胞减少、少尿血尿及管型尿,最终发生昏迷抽搐而死亡。但我们在抢救磷化锌中毒的临床实践中发现,磷化锌中毒患者因脑水肿而死亡是一个主要原因,而予以适当的脱水治疗往往可获得好转。这是因为,磷化锌是一种“亲神经毒物”。口服后在胃内与胃酸作
Jiangsu Medical Editorial Department: read your magazine in 1980 No. 2 “readers, authors, editors” column published by Nanjing Medical College Hu Jiantang on “zinc phosphide in the human body and how to rescue the process of” questions to answer, benefit from non shallow. As the authors point out, the clinical manifestations of oral zinc phosphide oral main clinical manifestations of oral mucosa and throat erosion, dry mouth, abdominal discomfort, speechless, nausea and vomiting diarrhea, hepatomegaly, jaundice, liver pain, systemic bleeding , Leukopenia, oliguric hematuria and tubular urine, the final occurrence of coma and death. However, in our clinical practice of rescuing zinc phosphide poisoning, we found that death from cerebral edema is a major cause of zinc phosphide poisoning in patients, and appropriate dehydration therapy can often be improved. This is because zinc phosphide is a “neurotropic agent.” After oral administration in the stomach and stomach acid