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克山病基本病理改变在于心肌变质,崩溃、溶解以至于坏死,病灶多是沿着小血管分布的,但血管并未阻塞,而且病理过程的开始是营养不良性的病变,可见变质是与营养和缺氧有关的。换句话说,和心肌梗死极相类似,动脉血带氧不足。根据我们千余例的观察,红血球带氧机能的确发生了障碍,其原因可能就是克山病的主因,正在研究中,我们初步认为高度的缺氧是可以解释克山病中心肌病变的。而且心肌变质过程一经开始后,其本身即可成为缺氧的主因,从而使变质过程愈趋恶化,这是无疑的。因为溶解与崩
Keshan disease is the basic pathological changes in myocardial deterioration, collapse, dissolution and even necrosis, lesions are mostly distributed along the small blood vessels, but the blood vessels are not blocked, and the pathological process of malnutrition is the beginning of the disease, it can be seen that deterioration and nutrition And hypoxia related. In other words, similar to myocardial infarction, arterial oxygen is not enough. According to our observations of more than a thousand cases, red blood cells with oxygen function does indeed occur obstacles, the reason may be the main cause of Keshan disease, is under study, we initially believe that a high degree of hypoxia can explain Keshan disease in the myopathy. And after the beginning of the process of myocardial deterioration, it can become the main cause of hypoxia, thus worsening the deterioration process, which is beyond doubt. Because of dissolution and collapse