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近十余年以来,对于心脏的生理学基础及临床研究有了飞速进展,对心功能不全的认识,也有所改观。过去认为心力衰竭乃为心脏本身的问题,其治疗上强烈主张应用强心药,以增加心肌收缩力,改善泵的功能,而忽视了周围血管状态对心功能的影响。1969年Gould证明了在心力衰竭时由于心搏出量降低,刺激了交感神经,产生代偿性周围血管反射性收缩,以提高血压,暂时地增加周围组织与重要脏器的灌注。如
In the past ten years or so, there has been rapid progress in the physiological basis and clinical researches of the heart, and the awareness of cardiac insufficiency has also changed. In the past that heart failure is the heart itself, the treatment is strongly advocated the use of cardiotonic drugs to increase myocardial contractility and improve the function of the pump, while ignoring the impact of peripheral vascular status on cardiac function. In 1969 Gould proved that in heart failure due to reduced cardiac output, stimulate the sympathetic, resulting in compensatory peripheral vascular reflex contraction to increase blood pressure, temporarily increase the surrounding tissue and vital organs perfusion. Such as