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对于慢性根尖肉芽肿的致病原因有各种不同的解释。细菌在这些组织中的存在可以解释这种病理现象。但是,一些研究则显示根尖的这种损害是无菌的。近年来,从免疫学观点对根尖肉芽肿进行的研究,证明这些组织中有各种免疫球蛋白和补体存在。因此,认为根尖肉芽肿的形成是一种免疫现象,其抗原物质,现代研究证实是存在于慢性牙髓炎根管内的内毒素。但是,尚未报道证实内毒素存在于根尖组织。本文研究结果表明,75%的慢性根尖肉芽肿组织中存在内毒素,说明根尖肉芽肿与内毒素有密切关系。根尖周损害的组织标本共30个,来源于没有明显牙周病史的牙齿,经根尖切除手术时获得。经组织学检查,分为炎症性(根尖肉芽肿)和非炎症性(瘢痕和囊肿)两种。全部试验过程都是在无热源(无内毒素)的条件下进行的,器械和玻璃器皿要经过至少
There are various explanations for the causative cause of chronic apical granuloma. The presence of bacteria in these tissues explains this pathology. However, some studies show that root damage is aseptic. In recent years, studies on apical granuloma from an immunological point of view have demonstrated the presence of various immunoglobulins and complement in these tissues. Therefore, it is believed that the formation of apical granuloma is an immune phenomenon, the antigenic material, modern studies have confirmed that there is endotoxin in the root canal of chronic pulpitis. However, no evidence has been reported that endotoxin is present in apical tissues. The results of this study show that 75% of chronic apical granuloma tissue endotoxin exist, indicating that there is a close relationship between apical granuloma and endotoxin. Periapical tissue damage in a total of 30 specimens from the absence of a clear history of periodontal history of teeth obtained by apical resection surgery. After histological examination, divided into inflammatory (apical granuloma) and non-inflammatory (scar and cyst) two. All testing is done without heat (endotoxin-free), and instruments and glassware go through at least