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患肺心病肺脑症时,由于慢阻肺引起的氧合障碍或肺泡通气不足影响气体交换,导致低氧血症和高碳酸血症,形成Ⅱ型呼衰。救治中除抗炎,解痉排痰,低流量吸氧等措施外,提高 PaO_2改善血氧供应,及排出 CO_2降低 PaCO_2和高碳酸血症,解除脑细胞 CO_2麻醉,是抢救病人生命的关键。为此本
Pulmonary heart disease pulmonary disease, due to chronic obstructive pulmonary oxygen barrier or alveolar hypoventilation affect gas exchange, leading to hypoxemia and hypercapnia, the formation of type Ⅱ respiratory failure. In addition to anti-inflammatory treatment, antispasmodic expectoration, low flow oxygen and other measures, to improve PaO_2 to improve blood oxygen supply, and the discharge of CO_2 reduce PaCO_2 and hypercapnia, relieve cerebral cell CO_2 anesthesia, is the key to saving the lives of patients. For this