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动脉瘤性蛛网膜下腔出血 (ＳＡＨ)后脑血管痉挛(ＣＶＳ)引起的迟发性缺血性神经功能障碍 ,大大增加了此病的死亡率和致残率。虽然近 30年来对脑血管痉挛的发生机制进行了广泛的实验和临床研究 ,但其确切的发病机制尚不明确[1] 。本文以观察动脉瘤性ＳＡＨ患者血液及脑脊液中Ｅ
Delayed ischemic neurological dysfunction caused by cerebral vasospasm (CVS) after aneurysmal subarachnoid hemorrhage (SAH) greatly increases the mortality and morbidity of the disease. Although the mechanism of cerebral vasospasm has been extensively studied experimentally and clinically in the past 30 years, its exact pathogenesis is not clear [1]. In this paper, to observe the aneurysmal SAH patients with blood and cerebrospinal fluid E