论文部分内容阅读
本文主要讨论羊膜腔及胎膜、脐带、胎盘、胎儿在妊娠期或分娩期发生的非特异性感染,目前称为羊膜腔感染综合征,好发于胎膜早破及产程延长者。胎膜有防御感染的功能,当该屏障受到破坏时容易发生羊膜腔感染综合征。其发生率随破膜-临产时问及破膜-分娩时间的延长而运渐增高。有资料表明,破膜后24小时以内分娩者,胎膜感染率不足10％,超过24小时者则在30％以上。Gosselin(1975年)统计了破膜-分娩时间与羊水细菌培养的关系(表1)。此外,还需重视胎膜感染与胎龄、胎儿体重的关系。有资料报道,体重等于或大于2 500克者,胎
This article discusses the amniotic and fetal membranes, umbilical cord, placenta, fetal non-specific infection during pregnancy or childbirth, is now known as amniotic cavity infection syndrome, occurs in premature rupture of membranes and labor extension. Membranes have the function of defensive infection, when the barrier is damaged prone to amniotic infection syndrome. The incidence of rupture of membranes with the time of labor and rupture of membranes - delivery time increased gradually. Data show that within 24 hours after rupture of the childbirth, the fetal infection rate of less than 10%, more than 24 hours in more than 30%. Gosselin (1975) analyzed the relationship between rupture of membrane and labor time and amniotic fluid bacterial culture (Table 1). In addition, the need to pay attention to the relationship between fetal membrane infection and gestational age, fetal weight. Data have been reported, weighing 2 500 grams or more who, fetal