论文部分内容阅读
对人拔出牙进行组织病理学观察,静止龋损的主要结构特征为表面区域完全矿化以及明显的牙本质小管酸化。除牙本质小管外的管间牙本质被充分矿化直至牙根表面,近牙本质表面的牙本质小管为无活性微生物或不同形状的磷灰石晶体所填塞。牙本质小管的酸化以发生在小管酸化带典型区域的三种管内矿化形式为其特征。矿化形式取决于结晶体的种类以及它们与有机结构如胶原纤维或造牙本质细胞突的相关关系。作者推测静止损害是由于以下原因而致: ①干扰牙髓基质扩散到入侵细菌(微生物)的内层屏障的形成;②由阻止细菌代谢产物进入牙本质的致密
Histopathology was performed on human teeth, and the main structural features of resting carious lesions were complete mineralization of the surface area and significant dentin tubule acidification. In addition to the dentinal tubules, the interphalangeal dentin is fully mineralized to the root surface, the dentinal tubules on the near dentin surface are filled with inactive microorganisms or different shapes of apatite crystals. Acidification of dentinal tubules is characterized by three in-tube mineralized forms that occur in the typical area of tubule acidification zone. The mineralization forms depend on the type of crystals and their correlation with organic structures such as collagen fibers or dentin. The authors speculate that resting damage is caused by: (1) interfering with the diffusion of the dental pulp matrix into the formation of the inner barrier of invading bacteria (microbes); (2) by inhibiting the formation of dense bacterial metabolites into the dentin