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肺原性心脏病患者,常因呼吸道感染、心衰、电解质紊乱、氮质血症、血氨增高等因素,使原来已经有器质性损害的肺组织发生进一步的功能阻碍,使机体缺氧加重,导致低氧血症,二氧化碳潴留,继发高碳酸血症和呼吸性酸中毒。脑组织在内环境出现上述病理生理改变之下,功能紊乱而出现所谓肺性脑病。肺性脑病在临床表现上常有三个类型:1.脑功能兴奋型。2.脑功能抑制型。3.兴奋—抑制不定型。当患者表现为1.2型时,症状常有:烦躁、兴奋、骚动不安或癫痫样发作。这时除针对病因治疗外,如何恰如其分地应用镇静剂解决因上述症状而引使机体代谢增加,耗氧加多,缺氧加重的不利环节便成了重要的治疗手段。当肺脑出现时,慎重地选择既可安定镇静而又不影响呼吸的药物已成了临床工作者所注意的问
Patients with pulmonary heart disease, often due to respiratory tract infections, heart failure, electrolyte imbalance, azotemia, increased blood ammonia and other factors, so that there have been organic damage to the lung tissue further dysfunction occurs, so that the body hypoxia Increased, leading to hypoxemia, carbon dioxide retention, secondary hypercapnia and respiratory acidosis. Brain tissue appears in the internal environment under the pathophysiological changes, dysfunction and the emergence of the so-called pulmonary encephalopathy. Pulmonary encephalopathy in the clinical manifestations often have three types: 1. Brain function excited type. 2. Brain function inhibition. 3. Excitement - inhibition of uncertainty. When the patient is manifested as type 1.2, the symptoms are often: irritability, excitement, disturbances or epileptic seizures. At this time in addition to the cause of treatment, how to properly apply sedatives to solve the above symptoms caused by increased body metabolism, oxygen consumption increased, the adverse aspects of hypoxia has become an important treatment. When lung and brain appear, careful selection can be calm and stable without affecting the respiratory drug has become a clinician’s attention