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流行性出血热(EHF)发病机理目前尚不十分清楚,但多数学者认为:宿主免疫反应起主要作用,即病毒入侵后大量复制。复合物沉积在血管内皮和肾小球基底膜上,从而导致多发性病理损害。本病的基本病理改变是全身毛细血管和小血管的损伤,从而引起各系统、各脏器的病理改变及功能障碍。病程中,尤以少尿期或移行阶段常引起各种严重的并发症。本刊1988年第10期曾报道一组EHF并发症文章,本期再继续报道一组EHF并发症文章,以使读者对EHF的多系统、多脏器损伤有所了解。
The pathogenesis of epidemic hemorrhagic fever (EHF) is not yet clear, but most scholars believe that the host immune response plays a major role, that is, a large number of replication after virus invasion. Complex deposition on the vascular endothelium and glomerular basement membrane, resulting in multiple pathological damage. The basic pathological changes of the disease is systemic damage of the capillaries and small blood vessels, causing pathological changes and dysfunctions of various systems and organs. Course of disease, especially oliguria or transitional stage often cause a variety of serious complications. This issue of 1988 No. 10 has reported a group of EHF complications articles, this issue and then continue to report a group of EHF complications articles to enable readers to EHF multi-system, multiple organ injury to understand.