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子痫前期、胎儿生长受限、早产、未足月胎膜早破、胎盘早剥、羊水栓塞和胎死宫内等妊娠并发症都是在感染、炎症反应、血栓、内分泌、免疫反应、遗传与环境等因素综合作用下导致的临床终点,常常表现为一系列的产科综合征,其中一些妊娠并发症中血栓形成和凝血酶改变均有报道。而且,在这些产科综合征出现之前,母体血液循环中就可以检测到高浓度的凝血酶-抗凝血酶III复合物以及凝血和抗凝血因子的变化。本文将对正常妊娠和妊娠并发症的母体在母胎界面上的凝血改变进行评估,探讨凝血系统改变在妊娠并发症的产生、发展中的特点和作用。
Pre-eclampsia, fetal growth restriction, premature delivery, preterm premature rupture of membranes, placental abruption, amniotic fluid embolism and fetal complications of pregnancy are in the infection, inflammation, thrombosis, endocrine, immune response, genetic And the environment and other factors lead to the clinical end point, often manifested as a series of obstetric syndrome, some of the complications of pregnancy thrombosis and thrombin changes have been reported. Moreover, high concentrations of thrombin-antithrombin III complex and changes in coagulation and anticoagulation factors can be detected in maternal blood circulation before these obstetric syndromes appear. This article will assess the normal pregnancy and pregnancy complications maternal interface changes in the maternal and fetal coagulation to explore changes in the coagulation system in the occurrence of pregnancy complications, the development of features and role.