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发生慢性心衰是由于心肌收缩障碍,而心肌收缩则取决于肌细胞膜结构的状况。生物膜损害始动因素之一是脂过氧化作用的活化,导致心肌收缩调节系统功能障碍。作者研究各种慢性心衰病人血液脂过氧化产物含量和抗氧化酶活性。病人和方法检查Ⅰ～Ⅲ期慢性心衰96例,其中梗塞后和动脉硬化性心硬化49例,年龄35～60岁;风湿性心脏病47例,年龄20～60岁。为进行对照,还检查20～40岁健康人20例;无慢性心衰症状的缺血性心脏病15例和风湿性心脏病11例,年
Chronic heart failure occurs due to myocardial contractility, while myocardial contraction depends on the muscle cell membrane structure. One of the initiating factors of biofilm damage is activation of lipid peroxidation, resulting in dysfunction of the myocardial contractile regulation system. The authors studied a variety of chronic heart failure patients blood lipid peroxidation product content and antioxidant enzyme activity. Patients and methods of examination Ⅰ ~ Ⅲ 96 cases of chronic heart failure, of which 49 cases of infarction and atherosclerotic heart disease, aged 35 to 60 years; 47 cases of rheumatic heart disease, aged 20 to 60 years. For the control, but also check 20 to 40 years old 20 healthy subjects; no symptoms of chronic heart failure in 15 cases of ischemic heart disease and rheumatic heart disease in 11 cases, year