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观察２０例急性心肌梗塞患者血液流变性、血小板聚集与超氧化歧化酶（ＳＯＤ）等指标。结果表明急性心肌梗塞时血液流变性异常、血小板聚集增高、ＳＯＤ降低，Ｐ＜０．０１。提示：血液流变性异常和血小板聚集可导致急性、慢性心肌缺血的加重。急性心肌梗塞再灌注产生大量的氧自由基加重缺血心肌超微结构的损害，使心肌梗塞范围扩大，更易发生室速和室颤动。
Blood rheology, platelet aggregation and superoxide dismutase (SOD) were observed in 20 patients with acute myocardial infarction. The results showed that abnormal blood rheology, platelet aggregation and SOD decreased in acute myocardial infarction (P <0.01). Tip: abnormal blood rheology and platelet aggregation can lead to acute, chronic myocardial ischemia worse. Acute myocardial infarction reperfusion produces a large number of oxygen free radicals to aggravate ischemic myocardial ultrastructure damage, the scope of myocardial infarction to expand, more prone to ventricular tachycardia and ventricular fibrillation.