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体外实验表明山莨菪碱和7643具有清除超氧阴离子的作用。在体实验二者均可抑制大鼠全脑 缺血30分钟再灌15分钟脑组织丙二醛含量的升高,减轻蛋白含量的下降,并促进全脑缺血30分钟的大鼠神经病征和行为障碍的恢复。说明二者对脑缺血性损伤具有保护作用。
In vitro experiments showed that anisodamine and 7643 had the effect of scavenging superoxide anion. Both in vivo and in vivo experiments could inhibit the increase of malondialdehyde (MDA) and the decrease of protein content in brain tissue of rats after reperfusion for 30 minutes after reperfusion for 30 minutes and promote the neurological signs of rats with global cerebral ischemia for 30 minutes Recovery of behavioral disorders. That both have a protective effect on cerebral ischemic injury.