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重度有机磷中毒常可发生脑水肿,如果忽视脑水肿,而只强调阿托品和解磷定的应用,不注意采取降低颅内压及防止脑水肿发生的措施,则难以避免脑疝形成而导致病人死亡。有机磷中毒机制主要是人体吸收有机磷后,有机磷与体内胆碱酯酶迅速结合,失去催化水解乙酰胆碱的作用,大量乙酰胆碱在体内蓄积,引起一系列以乙酰胆碱为传导介质的神经
Severe organic phosphorus poisoning can often occur brain edema, neglect of cerebral edema, but only emphasized the application of atropine and phosphate solution, do not pay attention to reduce the intracranial pressure and prevent the occurrence of cerebral edema measures, it is difficult to avoid the formation of the brain herniation and cause the patient to die . Organic phosphorus poisoning mechanism is mainly absorbed by the body after the organic phosphorus, organic phosphorus and cholinesterase rapid combination of the body loses the role of catalytic hydrolysis of acetylcholine, a large number of accumulation of acetylcholine in the body, causing a series of acetylcholine-mediated nerve