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本文内容取自1982年10月瑞士Basel肝昏迷讨论会议的会议录,讨论会有世界著名的肝病专家参加。会议主席为Conn HO。五种肝昏迷综合征及其发病机理(Conn HO) 肝昏迷综合征有五种:1.门体分流性,常见于肝硬化,动脉血氨增高为其特点。2.遗传性尿素循环酶缺乏,为一种先天性高血氨疾病。3.Reye综合征,为获得性尿素循环酶缺乏的疾病,部分也以高血氨为其表现。4.暴发型肝衰竭,可由病毒、药物或毒物引起,除脑病外所有肝功能亦受损害。5.假性肝性脑病,为肝硬化患者受镇静剂、镇痛剂作用,二氧化碳麻醉或低血糖所致。这些综合征有脑病、扑翼样震颤、
This article was taken from the meeting of the Basel Hepospic Coma in Switzerland in October 1982, attended by world renowned liver disease experts. The chairman of the conference is Conn HO. Five kinds of hepatic coma syndrome and its pathogenesis (Conn HO) There are five hepatic coma syndrome: 1. Portal body shunt, common in cirrhosis of the liver, elevated arterial ammonia as its characteristics. 2 hereditary urea cycle enzyme deficiency, as a congenital high blood ammonia disease. 3. Reye syndrome, acquired urease, a lack of disease, some also to high blood ammonia for its performance. 4. Fulminant hepatic failure may be caused by viruses, drugs or poisons. All liver functions except encephalopathy are also impaired. 5. Pseudohepatic encephalopathy, for patients with cirrhosis sedatives, analgesics, carbon dioxide anesthesia or hypoglycemia. These syndromes have encephalopathy, flapping-wing tremor,