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本文报告哮喘患者血清肌酸磷酸激酶(CPK)活性增高,并分析其增高的原因。两组病例,第一组为门诊观察达16个月以上的12名慢性哮喘患者,第二组系急性发作的5名患者,均排除酒精中毒、肝、心、肌肉疾患。第一组患者以支气管舒张剂维持,必要时用皮质激素。每月门诊随访一次,定期作CPK、CPK同功酶测定及肺功能检查。第二组急性发作者均住院治疗,开始时静脉注射氨茶硷及氢化考的松,情况改善后予以常规的支气管舒张剂治疗。各例在住院期间每日作血气分析、肺功
This article reports the elevation of serum creatine phosphokinase (CPK) activity in asthmatic patients and analyzes their causes. Two groups of patients, the first group of 12 patients with chronic asthma over 16 months in outpatient observation, and the second group of 5 patients with acute exacerbations were excluded alcoholism, liver, heart and muscle disorders. The first group of patients with bronchodilators to maintain, if necessary, corticosteroids. Monthly outpatient follow-up once a regular CPK, CPK isozyme assay and lung function tests. The second group of acute exacerbation were hospitalized, beginning with intravenous aminophylline and hydrocortisone, the situation improved to conventional bronchodilator therapy. Each case during hospitalization for daily blood gas analysis, lung function