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自发性脑出血是一种急性脑占位性损害.人们以脑内注入血液或用微气球占位等方式进行实验性脑出血研究.大量脑出血时,ICP明显升高.血肿形成后,周围脑组织即发生缺血性损害,且持续存在,不能为早期清除血肿所预防.结构性缺血损害仅局限于血肿邻近脑组织.血肿周围出现一个酶功能紊乱区,分布与~(14)C碘化氨替比林(~(14)C IAP)放射自显影显示的缺血区域一致.同时还伴有脂质过氧化物(LPO)增高、过氧化物歧化酶(SOD)降低等生化改变.合理的药物治疗有助于改善病变过程.
Spontaneous intracerebral hemorrhage is an acute brain-based damage.People with intracerebral injection of blood or the use of micro-balloon space for experimental cerebral hemorrhage study.My cerebral hemorrhage, ICP was significantly increased.After the formation of hematoma, around Brain tissue is ischemic and persists, and can not be prevented by early hematoma removal. Structural ischemic damage is confined to adjacent brain tissue of the hematoma. An area of enzyme dysfunction around the hematoma is found that is distributed with ~ (14) C Autoimmune imaging showed that the ischemic area was consistent with amidopyrine iodide (~ (14) C IAP), accompanied by increased biochemical changes such as increased lipid peroxides (LPO) and reduced superoxide dismutase (SOD) Reasonable medication helps to improve the course of the disease.