论文部分内容阅读
气管内插管能导致声门下狭窄,尤以幼儿更甚,且处理困难,了解甚少。为阐明这类狭窄发展的特征,弄清所生瘢痕组织内的异位软骨究系创伤反应抑或错构瘤?进行了动物实验和患儿研究。用切割钻对20只4周龄兔声门下造成环形损伤,10只的损伤限于上皮及上皮下层,余均伤及软骨膜及环状软骨最内层组织,于24周龄兔成熟期时将之处死后,取声门下组织做成5μm厚组织切片,用苏木素-偶氮焰红(azophloxin)染色。发现声门下气道复以正常呼吸上皮、腺体及弹力膜消失,上皮下再生组织由
Endotracheal intubation can lead to subglottic stenosis, especially young children even worse, and difficult to handle, little understood. To elucidate the characteristics of this stenotic development, elucidate the traumatic response or hamartoma of the ectopic cartilage in the resulting scar tissue. Animal studies and pediatric studies were performed. Ring wounds were performed on 20 4-week-old rabbits under the supraglottic girth with a dicing drill, and 10 lesions were limited to the epithelium and the subepithelium, leaving the perichondrocytes and the innermost layers of the cricoid cartilage to be sacrificed at the maturity of the 24-week-old rabbit After taking the subglottic tissue made of 5μm thick tissue sections, stained with hematoxylin - azophloxin (azophloxin). Found that the subglottic airway normal breathing epithelial complex, gland and elastic membrane disappeared, subcutaneous tissue from