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水稻叶片接种稻瘟病菌后，在非亲和性组合稻叶（１３１小种接种稻叶）中，过氧羟基脂肪酸分解酶（ＬｉｐｉｄＨｙｄｒｏｐｅｒｏｘｉｄｅ－ＤｅｃｏｍｐｏｓｉｎｇＥｎｚｙｍｅ，简称ＬＨＤＥ）活性在接种３６ｈ后显著增加，６０ｈ增加到最大；在亲和性组合稻叶（００７小种接种稻叶）和健康稻叶中，ＬＨＤＥ活性在接种３６、４８和６０ｈ后无显著增加。接种后４８ｈ的稻叶中的ＬＨＤＥ活性分别是亲和性组合稻叶和健康稻叶中ＬＨＤＥ活性的４．３和６．０倍。通过ＤＥＡＥ－Ｓｅｐｈａｒｏｓｅ阴离子交换层析，接种后４８ｈ的稻叶中的ＬＨＤＥ分为ＬＨＤＥ１和ＬＨＤＥ２两个组分。ＬＨＤＥ１组分在健康稻叶、亲和性组合稻叶和非亲和性组合稻叶中变化不大，总活性分别为０．０８Ｕ、０．０９Ｕ和０．１０Ｕ；但ＬＨＤＥ２组分在非亲和性组合稻叶中显著增加，而在亲和性组合稻叶和健康稻叶中变化不大，总活性分别为８．７２Ｕ、１．３０Ｕ和１．９０Ｕ。由此说明ＬＨＤＥ２是侵染诱导性ＬＨＤＥ，可能参与水稻对稻瘟病的抗性反应。
After rice leaf inoculation with Magnaporthe grisea, the activity of Lipid Hydroxide-Decomposing Enzyme (LHDE) increased significantly at 36h after inoculation and increased at 60h in non-inbred rice (131 inoculated rice) To the maximum; LHDE activity did not increase significantly at 36,48 and 60 h after inoculation in the combination paddy rice (007 races inoculated rice leaves) and healthy rice leaves. The LHDE activity in rice leaves 48 h after inoculation was 4.3 and 6.0 times higher than that of LHDE activity in affinity-combined and healthy rice leaves, respectively. By DEAE-Sepharose anion exchange chromatography, LHDE in rice leaves 48h after inoculation was divided into two components LHDE1 and LHDE2. The LHDE1 component did not change much in healthy rice leaves, inbred rice and non-inbred rice, with total activities of 0.08U, 0.09U and 0.10U, respectively. However, And the combination of rice leaves increased significantly, but in affinity combination rice leaves and healthy rice leaves little change, the total activity were 8.72U, 1.30U and 1.90U. Thus, LHDE2 was induced by inducible LHDE, which may be involved in the resistance of rice to rice blast.