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大鼠重度失血性休克30min后,左心室内压的最大上升速率(+dp/dt max)和左心室收缩压(LVSP)都显著下降(P<0.01),而左心室舒张末压(LVEDP)却有所增高(P<0.05)。说明心肌的收缩功能和舒张功能均出现障碍,给少量淋巴液后(用量为0.5ml/100g体重)使休克时下降的左心室内压的最大上升速率和左心室收缩压显著回升(P<0.05),而增高的左心室舒张末压回落不明显,提示淋巴液可改善休克心肌的收缩功能障碍,但对低血容量情况下的心肌舒张功能无明显改善。
After 30 minutes of severe hemorrhagic shock in rats, the maximal rate of increase of left ventricular pressure (+ dp / dt max) and left ventricular systolic pressure (LVSP) decreased significantly (P <0.01), while left ventricular end diastolic pressure (LVEDP) But increased (P <0.05). This shows that myocardial systolic and diastolic dysfunction are obstacles. After a small amount of lymph fluid (0.5ml / 100g body weight), the maximal rate of rise of left ventricular pressure and left ventricular systolic pressure decreased significantly at shock (P <0.05 ), While the increase in left ventricular end-diastolic pressure did not drop significantly, suggesting that lymph can improve myocardial systolic dysfunction, but no significant improvement in myocardial diastolic function in the case of hypovolemia.