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Shapiro(1965年)等发现两例自发的低体温发作与胼胝体发育不良有关,以后亦有类似报道,认为并非胼胝体发育不良,而系体温调节障碍,但多合并有影响体温中枢的中线结构发育异常,病理学研究发现丘脑下部前方纤维增生及神经元丧失,实验提示这些纤维和散热中枢有联系。发作性体温过低可由几分钟至几小时,甚至可持续达1周以上,本文报道一例胼胝体发育不良在癫痫发作后出现体温过低和代谢失调。男性,60岁,11岁时即有癫痫发作史,79年发生癫痫持续状态,偶见右侧Babinski氏征阳性,体温和T_4检查正常,无何原因的低血色素性贫血,有轻度智力低下。本次在服药的情况下又出现短暂的大发作,常伴有肌僵直,持续24小时以上,静注安定和氯丙嗪无效,14天后又出现大发作,血压正常而有窦性心动过速,1小时后病情恶化,出现昏迷,体温过低(直肠温29℃),面色苍白,低血压(收缩
Shapiro (1965) found that two cases of spontaneous hypothermia were associated with dysplasia of the corpus callosum. Similar reports were later reported that it was not dysplasia of the corpus callosum, but rather that the body temperature was impaired. However, the combination of central hypothermic central nervous system development , Pathological studies found that the lower part of the hypothalamus in front of fibrous proliferation and neuronal loss, experiments suggest that these fibers and the cooling center. Paroxysmal hypothermia can be from minutes to hours, and can last for more than 1 week. Here we report a case of hypoplastic hypothyroidism and metabolic disorders following the onset of seizures in a case of dysplasia of the corpus callosum. Male, 60 years old, 11 years of age, there is a history of seizures, 79 years of status epilepticus, occasional Babinski’s positive sign on the right side, normal body temperature and T_4, no reason hypochromic anemia, mild mental retardation . This time taking the case of another brief episode, often accompanied by muscle stiffness, sustained more than 24 hours, intravenous stability and chlorpromazine ineffective, 14 days after another major episode, normal blood pressure and sinus tachycardia , 1 hour later the condition deteriorated, a coma, hypothermia (rectal temperature 29 ℃), pale, low blood pressure (contraction