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干槽症的病因是一值得探讨的课题,分两个主要的学派,第一种学派认为牙槽窝根本无血凝块存在,第二种学派的观点是假设最初血凝块形成,以后血凝块分解留下一个空牙窝,作者收集了大量文献,现就干槽症的病因作如下讨论。为证明第一种观点,努力寻求阻止血凝块形成的因素,因素中要么是齿槽骨正常解剖结构的变异,要么是齿槽骨病理改变,血供不足。而Birn证明下磨牙周围齿槽骨具有丰富的血供,硬化性骨炎与干槽症的形成似乎无关。含缩血管麻药的过量浸润会阻断血
The cause of dry socket disease is a topic worth exploring, divided into two main schools, the first school that there is no blood clots alveolar nest, the second school of thought is the assumption that the initial formation of blood clots, after blood The clot breaks down to leave an empty socket, and the author has collected a great deal of literature and is discussing the causes of dry socket as follows. In order to prove the first view, efforts are being made to find a factor that can prevent the formation of blood clots. The factors are either the normal alveolar bone anatomical structure variation or the alveolar bone pathological changes and insufficient blood supply. Birn proved that there was abundant blood supply to the alveolar bone around the molars. Sclerosing osteoarthritis did not seem to be related to the formation of dry socket. Excessive stenosis of blood vessels containing blood vessels can block the blood