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随着工业飞速发展,世界石棉产量迅猛增加。1877～1967年间增长了8万倍,1970～1974年石棉的消耗量从300万吨增至500万吨。因此,接触石棉尘的人和与石棉有关的疾病日益增多。吸入石棉尘可导致肺纤维化的发生,在16世纪已有报导。大规模的流行病学调查资料证实,石棉肺的发生与石棉尘浓度(剂量)间有密切关系。但是,如何引起肺纤维化的机理至今尚不清楚。多年来,为了解释石棉肺的发病机制,曾提出各种学说,但没有一个理论能满意地解释下述事实:在接触相同石棉粉尘浓度的工人中,为何只有部分工人发病?停止接触多年后,为何仍可发生石棉肺或继续
With the rapid industrial development, the world’s asbestos production increased rapidly. From 1877 to 1967 an increase of 80000 times, from 1970 to 1974 asbestos consumption increased from 3 million tons to 5 million tons. As a result, people exposed to asbestos and asbestos-related diseases are on the rise. Inhalation of asbestos dust can lead to pulmonary fibrosis, which has been reported in the 16th century. Large-scale epidemiological survey data confirmed that the occurrence of asbestosis and asbestos dust concentration (dose) are closely related. However, the mechanism of how to cause pulmonary fibrosis is still not clear. Over the years, various theories have been put forward in order to explain the pathogenesis of asbestosis, but none of them can satisfactorily explain the fact that only some workers are exposed to exposure to the same dust concentration of asbestos. After many years of cease of exposure, Why asbestos may still occur or continue