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自1950年最早叙述慢活肝以来,探索慢活肝的可靠预后标志很少成功。后来发现预后与病因、组织学和血清学变化有关系,慢活肝的经过受着病因学深刻影响。药物引起的慢活肝在停用有害药物后可以逆转。有“狼疮”现象的病人常常没有B 型肝炎感染的标志,推测非A 非B 型肝炎也不出现“狼疮”现象。那些明显不是病毒起因的慢活肝,用皮质酮可以有奇效。而HBsAg 阳性慢活肝比阴性的预后差。通过各种血清学检查来判断病人预后己被学者
Since 1950, the earliest narrative chronic active liver, to explore the slow prognosis of the liver prognostic indicators with little success. Later found that the prognosis and etiology, histology and serological changes have a relationship, the slow-living liver has been profoundly affected by the etiology. Drug-induced slow liver can be reversed after deactivation of harmful drugs. Patients with “lupus” often do not have a sign of hepatitis B infection and speculate that “non-hepatitis B” does not show “lupus.” Those who are clearly not the cause of the virus slow living liver, with corticosterone can have miraculous effects. The HBsAg-positive slow liver than the negative prognosis is poor. By a variety of serological tests to determine the prognosis of patients has been scholars