【摘 要】
：
目的:探讨电针抗抑郁治疗快速起效的海马神经元保护和发生机制.方法:SD大鼠32只随机分为正常组、模型组、药物组(盐酸氟西汀)和电针组,每组8只.采用孤养和长期中等强度未预知
【基金项目】
：
江苏省教育厅重点实验室开放课题;
论文部分内容阅读
目的:探讨电针抗抑郁治疗快速起效的海马神经元保护和发生机制.方法:SD大鼠32只随机分为正常组、模型组、药物组(盐酸氟西汀)和电针组,每组8只.采用孤养和长期中等强度未预知应激制备应激大鼠抑郁症模型,运用免疫组化法观察治疗7天时各组大鼠在海马CAl、CA3、DG各区BDNF 及TrkB阳性神经元的表达.结果:①抑郁症模型存在海马区BDNF、TrkB表达下降,表现为阳性神经元平均灰度值上升、目标总面积下降;②治疗第7天,电针组海马神经元的BDNF及受体TrkB平均灰度值下降,且显著低于药物组,尤以CA3区为甚(P
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