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鉴于肝硬化腹水病人合并原发性细菌性腹膜炎(SBP)的发生率达10～25%,而腹水总蛋白在新出现腹水的鉴别诊断方面的不足,对传统的诊断性腹穿须重新评价。腹膜炎腹水中与腹水蛋白成比例的调理素水平下降(调理吞噬活性在蛋白<1g/dl 时消失)及长期菌血症看来与 SBP 有关。早期诊断腹膜炎的传统方法是细胞计数加多形核细胞的绝对数(APMN=白细胞数×多形核细胞百分数÷100),而大于250个/μl,认为是感染的证据。但有作者发现一些无菌性腹水病人,其 APMN
In view of the incidence of cirrhotic patients with primary bacterial peritonitis (SBP) rate of 10 to 25%, and ascites total protein in the new differential diagnosis of ascites inadequate, the traditional diagnosis of abdominal perforation should be re-evaluated. Reduced levels of opsonin in peritonitis ascites, which are in proportion to ascites protein (opsonophagocytic activity disappears at <1 g / dl protein), and long-term bacteremia appear to be associated with SBP. The traditional method of early diagnosis of peritonitis is to count the number of cells plus the absolute number of polymorphonuclear cells (APMN = the number of leukocytes × the percentage of polymorphonuclear cells ÷ 100), while greater than 250 per μl, which is considered as evidence of infection. But some authors have found some aseptic ascites in patients with their APMN