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通过对患病和健康施氏鲟 (AcipenserschrenckiiBrandt)进行的比较解剖、组织病理学与细胞病理学、流行病学研究结果表明 ,患病施氏鲟无明显的外部症状 ,临床解剖所见肝脏病变严重 :肝淤血、坏死、糜烂。肝组织脂肪变性、水样变性明显 ,肝细胞核浓缩或淡染、变形、核仁裂解 ,内质网扩张 ,线粒体嵴融解、崩解 ,胞浆内大量脂滴和液泡并将细胞器排挤成团。脑组织水样变性致液化性坏死 ,脑细胞胞浆呈液化泡状 ,核变形、浓缩 ,细胞膜融解。肾组织发生局灶性病变 ,病变肾细胞胞浆内有脂滴和液泡 ,核变形、融解 ,线粒体嵴融解、崩解。其它器官、组织、细胞未见病变。研究认为肝组织为原发性、中毒性坏死 ,脑组织病变与肝组织病变有依赖性 ,脑坏死为继发性液化性坏死 ,肾脏病变亦为继发性局灶性病变。因脑坏死而致鲟鱼死亡。文中还探讨了致病原因和机理以及危害性。
According to the comparative anatomy, histopathology and cytopathology of the diseased and healthy Acipenserschrenckii Brandt, the results of epidemiological studies showed that there was no obvious external symptoms in the diseased Acipenser schrenckii and severe liver disease in the clinical anatomy Liver congestion, necrosis, erosion. Liver degeneration, water degeneration was obvious, the liver cell nuclear condensation or light staining, deformation, nucleolus cleavage, endoplasmic reticulum dilation, mitochondrial ridge melting, disintegration, a large number of lipid droplets and vacuoles within the cytoplasm and squeezed into organelles. Liquefactive necrosis caused by watery degeneration of brain tissue, cytoplasm of brain cells are liquefied vesicular, nuclear deformation, concentration, cell membrane melting. Focal lesions in renal tissue, lipid droplets and vacuoles in the cytoplasm of diseased renal cells, nuclear deformation, melting, mitochondrial cristae melt, disintegration. No other organs, tissues, cells, lesions. Study that the liver tissue is primary, toxic necrosis, brain tissue lesions and liver disease dependent, cerebral necrosis secondary to liquefaction necrosis, renal disease is also a secondary focal lesion. Sturgeon death due to brain necrosis. The article also explores the causes and mechanisms of pathogenesis as well as the dangers.