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临床上多种病因可导致脑水肿,而由此出现的颅内高压和脑疝的形成常为致死的重要原因。使用脱水剂是解除脑水肿的主要措施之一。然而使用脱水剂过程中亦可能出现水、电解质失衡至低钠血症及低钠性(稀释性)脑水肿,引起病情复杂化的情况,且多被忽视或将此误认为是原发病情加重,而继续使用脱水剂,造成恶性循环,甚至死亡。为探讨脑水肿与血钠的关系,我们在1984～1987年6月间对收治的260例脑水肿患儿进行了血钠测定,现将结果报告如下。
Clinically a variety of causes can lead to cerebral edema, and thus the emergence of intracranial hypertension and herniation often form an important cause of death. The use of dehydrating agent is one of the main measures to relieve cerebral edema. However, dehydration agents may also cause imbalance of water and electrolytes to hyponatremia and hyponatremia (dilutional) brain edema, which complicates the condition and is often neglected or mistaken as an aggravating primary disease , While continuing to use dehydrating agents, causing a vicious cycle, or even death. In order to explore the relationship between cerebral edema and serum sodium, we conducted a blood sodium test on 260 children with cerebral edema admitted between 1984 and June 1987, and the results are reported below.