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昆虫的越冬耐寒过程与糖酵解、磷酸己糖途径和抗冻保护性物质合成等一些中间代谢有关的酶有关。该文对松针瘿蚊Thecodiplosisjaponensis老熟幼虫 1998/ 1999越冬期间体内上述代谢酶活性的变化进行了研究。越冬期间体内糖原磷酸化酶活性明显地增加 ,糖酵解有关的酶 (己糖激酶、乳酸脱氢酶和醛缩酶 )活性较低 ,以保证更多的碳源 (糖原 )转化成海藻糖。越冬期间 ,体内葡萄糖 6 磷酸脱氢酶活性增高所产生的还原型烟酰胺腺嘌呤二核苷酸磷酸 (NADPH) ,可为细胞在亚低温状态下发挥正常功能以及体内抗冻保护性物质的合成提供还原动力 ,同时通过调节体内海藻糖酶活性来维持越冬期间较高含量的海藻糖和移除春季体内累积的过多的海藻糖。
Insect overwintering cold process and glycolysis, hexose phosphate pathway and antifreeze protective material synthesis and some intermediate metabolism related enzymes. In this paper, we studied the changes of the above metabolic enzyme activities in mature cercaria larvae of Thecodiplosis japonensis 1998/1999 overwintering. Glycogen phosphorylase activity is significantly increased during winter, with less glycolysis-related enzymes (hexokinase, lactate dehydrogenase and aldolase) in order to ensure more carbon source (glycogen) conversion into Trehalose. During the winter, the reduction of nicotinamide adenine dinucleotide phosphate (NADPH) produced by increased glucose 6-phosphate dehydrogenase activity in vivo results in the normal functioning of cells in hypothermia and the synthesis of in vivo antifreeze protective substances Provide reducing motility while maintaining the higher content of trehalose during winter and the excess trehalose accumulated in spring by regulating trehalase activity in vivo.