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与年龄有关的黄斑变性(AMD)系指视网膜中央外层进行性损伤。实验研究已证明强光首先损伤AMD的变性部位。本组病例表明太阳辐射与某种变质性变化面导致的AMD有关。高能可见光和紫外线光可以由光化学机制产生分子损伤。氧可促发自由基一系列反应(光动力学作用),使损伤加重。黑色素能起防护作用,抵制来自日光的损害。大量材料证实:人类视网膜的太阳辐射范围损伤、光凝损伤以及在明亮照明环境中延长曝光的损伤,均可发生在AMD的相同部位。晶体和黑色素有保护视网膜抵抗AMD的作用。
Age-related macular degeneration (AMD) refers to progressive damage to the central outer retina. Experimental studies have shown that glare first damages the degeneration site of AMD. The patients in this group showed that solar radiation was associated with AMD due to some metamorphic changes. High energy visible light and ultraviolet light can cause molecular damage by photochemical mechanisms. Oxygen can trigger a series of free radical reactions (photodynamic action), so that damage increased. Melanin can play a protective role against the damage from sunlight. Numerous materials confirm that the human retina’s solar radiation range damage, photocoagulation damage, and prolonged exposure to damage in a brightly lit environment can occur in the same area of AMD. Crystals and melanin protect the retina against AMD.