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梗塞范围是影响急性心梗(AMI)预后的一个重要因素.CK 及 CK-MB 的活性与梗塞范围有良好相关关系.已知梗塞时 C-反应蛋白(CRP)与 CK-MB 活性相关,但对其与梗塞大小罕有研究.作者发现血 CRP 在无 Q 波梗塞时并不升高.因此对能迅速获得心肌再灌注从而限制了 CRP 肝脏的合成,故认为 AMI 时成功的溶栓治疗应有类似效果,因而研究了链激酶治疗与否时的血 CRP浓度.
The infarct size is an important factor affecting the prognosis of acute myocardial infarction (AMI), and the activity of CK and CK-MB has a good correlation with the extent of infarction. C-reactive protein (CRP) is known to be associated with CK-MB activity at infarction Rarely studied with regard to infarct size, the authors found that blood CRP does not increase without Q-wave infarction, and therefore prompt myocardial reperfusion to limit the synthesis of CRP liver, so that successful thrombolytic therapy at AMI should have A similar effect was therefore investigated for the concentration of blood CRP when streptokinase is treated or not.