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在 Q-T 间期延长综合征和急性心肌梗塞病人,心肌复极异常与恶性心律失常的形成有关。睡眠系副交感张力增高或交感张力减弱的自然过程,可引起与心率减慢无关的 Q-T 间期延长。鉴于睡眠时副交感神经张力增高引起的心率减慢本身可延长 Q-T间期,从而掩盖交感神经的直接作用,本文报道用新的方法测定不从属于心率的 Q-T 间期,以证实人在睡眠时出现的 Q-T 间期延长。方法:15名受试者(11名男性,4名女性)作72～144小时连续心电图记录。所有受试者均符合下列标准:(1)病史、体检和静息心电图无心脏病证
In the Q-T interval prolongation syndrome and acute myocardial infarction, myocardial repolarization abnormalities are associated with the formation of malignant arrhythmias. The natural process of increased parasympathetic tone or reduced sympathetic tone in the sleep department can lead to prolongation of the Q-T interval that is unrelated to heart rate slowing. In view of the fact that slowing the heart rate due to increased parasympathetic tone during sleep prolongs the QT interval to mask the direct effects of sympathetic nerves, a new method for determining the QT interval that is not subordinate to heart rate is reported in this paper to confirm that humans appear during sleep QT interval prolongation. Methods: Fifteen subjects (11 males and 4 females) underwent continuous electrocardiographic recording for 72 to 144 hours. All subjects met the following criteria: (1) medical history, physical examination and resting ECG without heart disease card