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通过观察微量阿司匹林（Ｍ－ＡＳＡ）对血小板聚集、血栓形成、血液流变及凝血机制作用表明，Ｍ－ＡＳＡ不仅能明显抑制血小板释放ＴＸＡ２，显著降低血小板聚集率和血栓指数Ｑ值，而且可明显改善红细胞聚集性及变形能力。但对全血粘度、凝血酶原时间则无明显影响。可见虽然凝血机制、血液粘滞性与血栓形成有密切关系，但与血栓形成的独特机理无关。同时还表明血液的高粘滞性也并非都由凝血机制亢进所致。
By observing the effect of M-ASA on platelet aggregation, thrombosis, hemorheology and coagulation mechanism, M-ASA can not only significantly inhibit TXA2 release from platelets, but also significantly reduce platelet aggregation rate and Q value of thrombosis, Improve red blood cell aggregation and deformability. However, the whole blood viscosity, prothrombin time no significant effect. Shows that although the coagulation mechanism, blood viscosity and thrombosis are closely related, but has nothing to do with the unique mechanism of thrombosis. At the same time also shows that the high viscosity of the blood is not caused by hypercoagulability of the coagulation mechanism.