miR-1182 attenuates gastric cancer proliferation and metastasis by targeting the open reading frame

• 作 者: DanZhang;Shi-MingYang;
• 机 构: Department of Gastroenterology, Xinqiao Hospital, Third Military Medical University, Chongqing 40003
• 出 处: 第17届西南地区消化病学术会议暨2014贵州省消化病及消化内镜学术年会
• 发表日期: 2014年期

　　Background hTERT is an ideal anticancer target because it is universally expressed in cancer cells and plays a crucial role in carcinogenesis.In our previous study,we demonstrated that over-expression of miR-1182 suppresses hTERT expression.However,the mechanism of how miR-1182 modulates hTERT has not yet been investigated.