• 不 限
  • 期刊论文
  • 硕博论文
  • 会议论文
  • 报 纸
  • 英文论文
  2. 您的位置
  3. 首页
  4. 会议论文
  5. p38 MAPK participates in the up-regulation of GLT-1 during the induction of brain ischemic tolerance

p38 MAPK participates in the up-regulation of GLT-1 during the induction of brain ischemic tolerance

来源 :The 6th FAONS Congress and 11th Biennial Conference of CNS(第 | 被引量 : 0次 | 上传用户：helen_fu
【摘 要】
：
Our previous study has proved that the up-regulation of glial glutamate transporter 1(GLT-1)induced by cerebral ischemic preconditioning(CIP)played an important role in the acquisition of brain ischem
【作 者】
：
Min Zhang Jian-Xue Gong Jia-Lei Wang Meng-Yang Jiang Li Li Hang Zhao Yu-Yan Hu Wen-Bin Li 
【机 构】
：
Department of Pathophysiology,Hebei Medical University,361 Zhongshan East Road,Shijiazhuang,050017,P
【出 处】
：
The 6th FAONS Congress and 11th Biennial Conference of CNS(第
【发表日期】
：
2015年3期
【关键词】
：
p38 MAPK glial glutamate transporter-1 brain ischemic tolerance cerebral ischemi 
下载到本地 , 更方便阅读
下载此文 赞助VIP
声明 : 本文档内容版权归属内容提供方 , 如果您对本文有版权争议 , 可与客服联系进行内容授权或下架
论文部分内容阅读
　　Our previous study has proved that the up-regulation of glial glutamate transporter 1(GLT-1)induced by cerebral ischemic preconditioning(CIP)played an important role in the acquisition of brain ischemic tolerance in rats.
其他文献
Cerebral ischemia-induced occludin degradation promotes blood brain barrier endothelial cell apoptos
Intracerebral hemorrhage is the most dangerous complication in tPA thrombolytic therapy for ischemic stroke,which occurs as a consequence of endothelial cell apoptosis of the blood brain barrier(BBB)d
会议
Ischemic strokeblood brain barrieroccludinendothelial cellapoptosisthrombol
Role of autophagy in the cerebral ischemic reperfusion injury of mice and its cPKCγ-mediated molecul
To investigate the progression of autophage in the peri-infarct tissue after middle cerebral artery occlusion(MCAO)mice models; To explore the function of autophage and its possible molecular mechanis
会议
cerebral ischemia reperfusion injuryProtein Kinase C gamma(cPKCγ)peri-infarct
Methylene Blue improves the disruption of blood brain barrier and attenuates astroglia activation af
Methylene blue(MB)was first synthesized in 1876 and has been used as treatment for malaria,methemoglobinemia,and cyanide poisoning.
会议
methylene bluetransient middle cerebral artery occlusionblood-brain barrieras
A MRI study of methylene blue on ischemia/reperfusion-induced cerebral edema
The neuroprotective effect of methylene blue has been proved by a number of studies for its unique functions as an alternative electron carrier,as well as energy-enhancing and antioxidant agent.
会议
methylene bluestrokecerebral edemaMRI
von Willebrand factor contributes to poor outcome in a mouse model of intracerebral hemorrhage
Objective: von Willebrand factor(VWF)has recently been demonstrated to promote inflammation processes.In the present study,we investigated the pathophysiological role of VWF after experimental intrace
会议
blood-brain barrieredema formationinflammationintracerebral hemorrhageVWF
APE/Ref-1 mediates the prevention of 5-HMF against oxidative injury caused by brain ischemia in rats
Cerebral ischemia-reperfusion injury plays an important role in the evolution of brain damage through mechanisms that include the generation of reactive oxygen species(ROS),oxidative stress,inflammati
会议
5-HMFAPE/Ref-1ischemiaH2O2PC12ROS
Focal ischemia induces local neurogenesis from astrocytes in adult cortex
Background Adult cortex has long been regarded as “non-neurogenic”.Recent studies have indicated the occurrence of neurogenesis in cortex after ischemic stroke.
会议
neurogenesiscortexischemiaastrocyte
Role of interleukin-17 in cerebral ischemic injury and its molecular mechanism in mice with ischemic
To explore the dynamic change of interleukin-17A(IL-17A)in cerebrospinal fluid(CSF)and blood after ischemic stroke in vivo.Futher,to observe whether there are some nerve cells involved in the secretio
会议
interleukin-17ischemic strokeoxygen-glucose deprivation (OGD)
The pathway of MAGED1-miR-137-SIRT1 involved in ischemic hippocampal cell death in mice
Objective Hippocampus is one of the most vulnerable regions after ischemic stroke.Until now the mechanism of ischemia-reperfusion injury has not been understood clearly.
会议
ischemiahippocampusmiR-137SIRT1MAGED1
Up-regulated expression of NT-3 mediated by hypoxia response element attenuates cerebral ischemia/re
To investigate the neuroprotective effects of neurotrophin-3(NT-3) expression controlled by five copies of the hypoxia-responsive elements(5HRE)after focal cerebral ischemia.
会议
hypoxia-inducible gene expressionneuroprotectionischemic strokegene therapy