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目的:探讨姜黄素对放线菌素D(ActD)/TNF-α协同诱导PC12细胞凋亡的作用机制,为防治HIV相关认知障碍及相关疾病提供实验依据。方法:MTT法测定姜黄素及ActD/TNF-α损伤PC12细胞的最佳剂量;流式细胞术、RT-PCR等测细胞
Aims: To investigate the mechanism of curcumin on PCD apoptosis induced by actinomycin D (ActD) / TNF-α and to provide experimental evidence for the prevention and treatment of HIV-related cognitive disorders and related diseases. Methods: The best dosage of curcumin and ActD / TNF-α in PC12 cells were determined by MTT assay. Flow cytometry, RT-PCR and other test cells