论文部分内容阅读
Objective: Previous experiments showed that mdivi-1 (Mitochondrial division inhibitor) protects brain from ischemia/reperfusion injury (IRI) in rats followed by transient cerebral ischemia, but the precise mechanisms of its neuroprotective effect are far from clear.This study aims to elucidate the effect of mdivi-1 on energy metabolism and neuronal apoptosis induced by I/R.