TMEM16A and myocardin form a positive feedback loop that isdisrupted by KLF5 during Ang Ⅱ induced va

来源 :中国生物化学与分子生物学会第十一次会员代表大会暨2014年全国学术会议 | 被引量 : 0次 | 上传用户：zql0913

【摘要】

：

【作者】

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Xin-hua Zhang

【机构】

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Key Laboratory of Neural and Vascular Biology, Department of Biochemistry and Molecular Biology,Mini

【出处】

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中国生物化学与分子生物学会第十一次会员代表大会暨2014年全国学术会议

【发表日期】

：

2014年3期

【关键词】

：

TMEM16A myocardin feedback loop Kruppel-like factor 5 vascular smoothm uscle cel

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TMEM16A protein is recently identified as an important component of Ca+-dependentC1-channels (CaCCs) in vascular smooth muscle cells (VSMCs).However, the functions ofrMEM16A protein and the mechanisms responsible for regulating TMEM16A gene expressionin the vessels remain largely unclear.

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TMEM16A and myocardin form a positive feedback loop that isdisrupted by KLF5 during Ang Ⅱ induced va

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