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  5. αCaMKII overexpression impairs behavioral flexibility and NMDAR-dependent LTD in the medial prefront

αCaMKII overexpression impairs behavioral flexibility and NMDAR-dependent LTD in the medial prefront

来源 :The 6th FAONS Congress and 11th Biennial Conference of CNS(第 | 被引量 : 0次 | 上传用户：al035258
【摘 要】
：
Objective The medial prefrontal cortex(mPFC)participates in the behavioral flexibility.As a major downstream molecule in the NMDA receptor signaling,alpha-Ca2+/calmodulin-dependent protein kinase II(α
【作 者】
：
Jing Ma Yan-Hong Duan Zhen-Xia Qin Jia-Yue Wang Mei-Chen Xu Si-Qi Zhou Xiao-Hua Cao 
【机 构】
：
Key Laboratory of Brain Functional Genomics,MOE & STCSM,East China Normal University,Shanghai,200062
【出 处】
：
The 6th FAONS Congress and 11th Biennial Conference of CNS(第
【发表日期】
：
2015年3期
【关键词】
：
αCaMKII behavioral flexibility long-term depression medial prefrontal cortex AMP 
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　　Objective The medial prefrontal cortex(mPFC)participates in the behavioral flexibility.As a major downstream molecule in the NMDA receptor signaling,alpha-Ca2+/calmodulin-dependent protein kinase II(αCaMKII)is crucial for hippocampal long-term potentiation(LTP)and hippocampus-related memory.
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